Seborrheic keratoses represent the most prevalent benign growths in older individuals. While their precise origin remains a mystery, it is believed that they stem from the uncontrolled expansion of mutated epidermal keratinocytes.
Histologically, seborrheic keratoses display clear signs of proliferation.
In some instances, they are inherited via autosomal dominant pattern.
Sunlight exposure appears to contribute to the development of certain seborrheic keratoses.
Typically, these growths are devoid of symptoms, but they can become bothersome.
Lesions may provoke itching, irritation from clothing friction, and subsequent inflammation.
Furthermore, these growths are often considered aesthetically displeasing and can have negative psychological associations.
While lesions may occasionally resolve spontaneously, this is not a common occurrence.
The onset of seborrheic keratoses is typically marked by the emergence of one or more well-defined, light brown, flat macules.
These lesions can appear in limited numbers or be quite numerous.
Initially, they display a velvety or finely warty surface, later progressing to an uneven texture with numerous plugged follicles.
Their distinctive feature is an appearance of being stuck-on to the skin surface.
The pigmentation of these growths can range from pale brown with pink undertones to dark brown or even black.
Their natural course involves gradual enlargement, increased thickness, and the gradual development of new lesions.
A range of methods can be employed to treat seborrheic keratoses.
These include cryotherapy using carbon dioxide or liquid nitrogen, electrodesiccation, electrodesiccation followed by curettage, curettage alone, shave biopsy or excision using a scalpel, or the utilization of a laser or dermabrasion surgery.

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